Impact of Artificial Sweeteners on the GI Microbiota
Student’s First name, middle initial(s), last name.
Institution Affiliation
Course Number and Name
Instructor Name
Date
Impact of Artificial Sweeteners on the GI Microbiota
I agree with your post on the evidence surrounding the impact of artificial sweeteners on the GI microbiota. This is because artificial sweeteners, or non-nutritive sweeteners, are commonly used as sugar substitutes because they are sweet like sugar, but at greater intensities, and do not provide calories (Bian, 2017). Stevia, acesulfame potassium, saccharin, aspartame, and sucralose are some of the examples of artificial sweeteners, as illustrated in your post. Moreover, analysts accept that manufactured sweeteners might be changing our intestine microbiota, leading to glucose narrow mindedness in people who expend these items (Stichelen, Rother, & Hanover, 2019). You have shown that people have been eating artificial sweeteners for a long time now since they are cheap. Furthermore, one of your research study has shown P < 0.05 increased Firmicutes significance in mice compared to mice. According to research, mice devouring artificial sweeteners created glucose bigotry, but mice devouring sugar or plain water did not create glucose narrow mindedness.
Consequently, according to your analysis, sucralose reduced the body weight of mice fed normal chow (P < 0.0001), but not those given a higher fat diet despite no changes in calorie intake between groups. Sucralose also significantly increased fecal output in both the normal chow group and the high fat diet group (P < 0.05) (Bian, 2017). Another research in your post analyzed the effect of sucralose on the inflammation and microbiota mediators in mice. These studies illustrate that artificial sweeteners, such as sucralose, can affect the intestine microbiota and seem impact metabolic components like weight and irritation. Whereas artificial sweeteners are regularly suggested for weight misfortune, specialists ought to note the results that will occur from expanding utilization of these choices (Stichelen, Rother, & Hanover, 2019). However, to add to your discussion, the colonic microbiota may be a compounded milieu that plays a central part in controlling numerous physiological forms. Thus, I believe that your post is on point with a vivid discussion of the impact of artificial sweeteners on the GI microbiota.
References
Bian, X., Chi, L., Gao, B., Tu, P., Ru, H., & Lu, K. (2017). Gut Microbiome Response to
Sucralose and its Potential Role in Inducing Liver Inflammation in Mice. Frontiers in
Physiology, 8, 487.
Stichelen, O. V., Rother, K. I., & Hanover, J. A. (2019). Maternal Exposure to Non-Nutritive
Sweeteners Impacts Progeny’s Metabolism and Microbiome. Frontiers in
Microbiology, 10, 1360.