Obstructive Sleep Apnea

Obstructive Sleep Apnea

Obstructive sleep apnea (ASA) has advanced implications on various body organs of the body. The effects, in return, have varying relevance to causing cardiovascular disease. Hence, ASA is frequently answerable to multiple diseases such as diabetes, systematic inflammation, dyslipidemia, deposition of fats in the visceral area, and high blood pressure. Due to this fact, researchers in this discipline carried out a two-in-one research study to examine the interactions that exist between obstructive sleep apnea and obesity. This paper seeks to provide a brief description of these research studies in the light of hypothesis, outcomes, and conclusions.

The first research study is by Corral et al. (2010) titled “Interactions between obesity and obstructive sleep apnea: implications for treatment.” It hypothesizes that obstructive sleep apnea evolves rapidly, and that is it responsible for a significant number of cardiovascular problems due to negative implications on organs. Indeed, OSA, according to the researchers, results in hypertension’s etiology. The reality is besides other known medical conditions like diabetes and heart failure. Nevertheless, they admit that there is a need for further research to determine whether or not OSA is casually liable to cause diseases. The first research achieved its aim by providing facts and statistics to prove its hypothesis. For example, it claims that obesity cases linked with OSA range from 25%-45%. Due to an increase in OSA cases, there has been a rise in obesity cases too.

Further analysis from this research indicates that 60% of the total population in developed countries is obese. OSA is known to cause sleep disruptions, which further threaten sleep mechanisms, this increasing complexity of treatment. This withstanding, too much fat in the body affects the functioning of organs such as the heart. In concluding this research, the researchers noted that there are severe consequences of mitigating cardiometabolic health problems caused by both obesity and OSA (Yaeger, Cash, Parham, Frankel, Deo, Schaller, … & Riley, 2018). Therefore, weight reduction is a recommended option for countering this concern.

Another study by Angela Adelizzi (2020) published in the Obesity Medicine Association to address the relationship between obesity and sleep Apnea drives to an almost similar conclusion as one above. Just like the one conducted by Corral et . (2010), Adelizzi argues that OSA affects many body parts negatively and is caused by factors such as hypertension, insulin resistance, and fat deposition. The hypothesis, therefore, is to examine the relationship between OSA and the said health challenges. Their study is perfect since it provides facts and figures to prove the original hypothesis. For instance, Adelizzi believes that up to 25% to 45% of the population is on the verge of obesity. Also, patients with these conditions already existing in them and who go-ahead to add above 10% of the present body weight increase the probability of disease worsening by six-folds.

Conversely, a 20% decline in weight means a high chance of improvement in health. Unlike the previously discussed study, this study insists on the contribution of glucose intolerance to obstructive sleep apnea. Also, abnormalities in lipid regulation within the human body is another issue that is a trigger to OSA.

References

Angela Adelizzi, DO (2020). Obesity and Obstructive Sleep Apnea. Obesity Medicine Association. https://obesitymedicine.org/obesity-and-sleep-apnea

Romero-Corral, A., Caples, S. M., Lopez-Jimenez, F., & Somers, V. K. (2010). Interactions between obesity and obstructive sleep apnea: implications for treatment. Chest, 137(3), 711-719.

Yaeger, A., Cash, N. R., Parham, T., Frankel, D. S., Deo, R., Schaller, R. D., … & Riley, M. P. (2018). A Nurse‐led limited risk factor modification program to address obesity and obstructive sleep apnea in atrial fibrillation patients. Journal of the American Heart Association, 7(23), e010414.